DOUBLE-JABBED Scots are now more likely to be admitted to hospital with Covid than the unvaccinated amid an increase in elderly people falling ill due to waning immunity.
But we must tax the unvaxxed since they are spreading the disease, right...
DOUBLE-JABBED Scots are now more likely to be admitted to hospital with Covid than the unvaccinated amid an increase in elderly people falling ill due to waning immunity.
Most Covid jab ‘side effects’ are not caused by vaccines – study
Many people are so worried about Covid vaccine ‘side effects’ they actually feel them even if they get placebo, new research claims
Vast majority of Covid vaccine ‘side effects’ are caused by people's expectations rather than by the vaccine, Harvard Medical School researchers say, after analyzing the reports of more than 45,000 trial participants.
Various “systemic” side effects, such as headaches, tiredness, and joint pain were reported in both halves of the focus group: among those who received various Covid vaccines, as well as those who unknowingly received placebo.
After analyzing the reports, scientists from the Boston-based Beth Israel Deaconess Medical Center came to the conclusion that the so-called “nocebo effect” – unpleasant sensations caused by anxiety or bad expectations – accounted for three quarters of all reported vaccine side effects.
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Mir wrote:AND how about this new "study"!
https://www.rt.com/news/546445-vaccines-nocebo-research-effects/
Most Covid jab ‘side effects’ are not caused by vaccines – study
Many people are so worried about Covid vaccine ‘side effects’ they actually feel them even if they get placebo, new research claims
Vast majority of Covid vaccine ‘side effects’ are caused by people's expectations rather than by the vaccine, Harvard Medical School researchers say, after analyzing the reports of more than 45,000 trial participants.
Various “systemic” side effects, such as headaches, tiredness, and joint pain were reported in both halves of the focus group: among those who received various Covid vaccines, as well as those who unknowingly received placebo.
After analyzing the reports, scientists from the Boston-based Beth Israel Deaconess Medical Center came to the conclusion that the so-called “nocebo effect” – unpleasant sensations caused by anxiety or bad expectations – accounted for three quarters of all reported vaccine side effects.
Probably the vast majority of people that took the jab in the first place - fully expected this "scientific miracle" to save them from the nasty killer virus. Now if the above applies why do they keep on getting covid?
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A new report from the federal Centers for Disease Control and Prevention shows that in 94 percent of the
cases of those who died from COVID-19, another condition — in most cases likely brought on or exacerbated by
the presence of the COVID virus — was also at work on the victim.
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Fourth Pfizer Dose Is Insufficient to Ward Off Omicron, Israeli Trial Suggests
A fourth dose of the Pfizer-BioNTech vaccine was insufficient to prevent infection with the omicron variant of Covid-19, according to preliminary data from a trial in Israel released Monday.
Two weeks after the start of the trial of 154 medical personnel at the Sheba Medical Center in Tel Aviv, researchers found the vaccine raised antibody levels.
The lunatics have taken over the asylum.
Endogenous Deficiency of Glutathione as the Most Likely Cause of Serious Manifestations and Death in COVID-19 Patients
Higher rates of serious illness and death from coronavirus SARS-CoV-2 (COVID-19) infection among older people
and those who have comorbidities suggest that age- and disease-related biological processes make such individuals more sensitive to
environmental stress factors including infectious agents like coronavirus SARS-CoV-2. Specifically, impaired redox homeostasis and
associated oxidative stress appear to be important biological processes that may account for increased individual susceptibility to
diverse environmental insults. The aim of this Viewpoint is to justify (1) the crucial roles of glutathione in determining individual
responsiveness to COVID-19 infection and disease pathogenesis and (2) the feasibility of using glutathione as a means for the
treatment and prevention of COVID-19 illness. The hypothesis that glutathione deficiency is the most plausible explanation for
serious manifestation and death in COVID-19 patients was proposed on the basis of an exhaustive literature analysis and
observations. The hypothesis unravels the mysteries of epidemiological data on the risk factors determining serious manifestations of
COVID-19 infection and the high risk of death and opens real opportunities for effective treatment and prevention of the disease.
Severe Glutathione Deficiency, Oxidative Stress and Oxidant Damage in Adults Hospitalized
with COVID-19: Implications for GlyNAC (Glycine and N-Acetylcysteine) Supplementation
Humanity is battling a respiratory pandemic pneumonia named COVID-19 which has
resulted in millions of hospitalizations and deaths. COVID-19 exacerbations occur in waves that
continually challenge healthcare systems globally. Therefore, there is an urgent need to understand
all mechanisms by which COVID-19 results in health deterioration to facilitate the development
of protective strategies. Oxidative stress (OxS) is a harmful condition caused by excess reactive-
oxygen species (ROS) and is normally neutralized by antioxidants among which Glutathione (GSH)
is the most abundant. GSH deficiency results in amplified OxS due to compromised antioxidant
defenses. Because little is known about GSH or OxS in COVID-19 infection, we measured GSH,
TBARS (a marker of OxS) and F2-isoprostane (marker of oxidant damage) concentrations in 60 adult
patients hospitalized with COVID-19. Compared to uninfected controls, COVID-19 patients of all age
groups had severe GSH deficiency, increased OxS and elevated oxidant damage which worsened
with advancing age. These defects were also present in younger age groups, where they do not
normally occur. Because GlyNAC (combination of glycine and N-acetylcysteine) supplementation has
been shown in clinical trials to rapidly improve GSH deficiency, OxS and oxidant damage, GlyNAC
supplementation has implications for combating these defects in COVID-19 infected patients and
warrants urgent investigation.
Effects of Basic Amino Acids and Their Derivatives on SARS-CoV-2 and Influenza-A Virus Infection
Amino acids have been implicated with virus infection and replication. Here, we demonstrate the effects of two basic amino acids, arginine and lysine, and their ester derivatives on infection of two enveloped viruses, SARS-CoV-2, and influenza A virus. We found that lysine and its ester derivative can efficiently block infection of both viruses in vitro. Furthermore, the arginine ester derivative caused a significant boost in virus infection. Studies on their mechanism of action revealed that the compounds potentially disturb virus uncoating rather than virus attachment and endosomal acidification. Our findings suggest that lysine supplementation and the reduction of arginine-rich food intake can be considered as prophylactic and therapeutic regimens against these viruses while also providing a paradigm for the development of broad-spectrum antivirals.
Glycyrrhizin Effectively Inhibits SARS-CoV-2 Replication by Inhibiting the Viral Main Protease
The outbreak of SARS-CoV-2 developed into a global pandemic affecting millions of people worldwide. Despite one year of intensive research, the current treatment options for SARS-CoV-2 infected people are still limited. Clearly, novel antiviral compounds for the treatment of SARS-CoV-2 infected patients are still urgently needed. Complementary medicine is used along with standard medical treatment and accessible to a vast majority of people worldwide. Natural products with antiviral activity may contribute to improve the overall condition of SARS-CoV-2 infected individuals. In the present study, we investigated the antiviral activity of glycyrrhizin, the primary active ingredient of the licorice root, against SARS-CoV-2. We demonstrated that glycyrrhizin potently inhibits SARS-CoV-2 replication in vitro. Furthermore, we uncovered the underlying mechanism and showed that glycyrrhizin blocks the viral replication by inhibiting the viral main protease Mpro that is essential for viral replication. Our data indicate that the consumption of glycyrrhizin-containing products such as licorice root tea of black licorice may be of great benefit for SARS-CoV-2 infected people. Furthermore, glycyrrhizin is a good candidate for further investigation for clinical use to treat COVID-19 patients.
kvs wrote:
I applaud the UK for moving back to sanity. By contrast, Kanada is going off the rails as our SJW faggot prime minister is imposing more
and more mandates.
Full blown certifiable psychopaths. Perhaps someone should develop a "vaccine" for that disease.
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SARS-CoV-2 infection induces long-lived bone marrow plasma cells in humans
Long-lived bone marrow plasma cells (BMPCs) are a persistent and essential source of protective antibodies1,2,3,4,5,6,7. Individuals who have recovered from COVID-19 have a substantially lower risk of reinfection with SARS-CoV-28,9,10. Nonetheless, it has been reported that levels of anti-SARS-CoV-2 serum antibodies decrease rapidly in the first few months after infection, raising concerns that long-lived BMPCs may not be generated and humoral immunity against SARS-CoV-2 may be short-lived11,12,13. Here we show that in convalescent individuals who had experienced mild SARS-CoV-2 infections (n = 77), levels of serum anti-SARS-CoV-2 spike protein (S) antibodies declined rapidly in the first 4 months after infection and then more gradually over the following 7 months, remaining detectable at least 11 months after infection. Anti-S antibody titres correlated with the frequency of S-specific plasma cells in bone marrow aspirates from 18 individuals who had recovered from COVID-19 at 7 to 8 months after infection. S-specific BMPCs were not detected in aspirates from 11 healthy individuals with no history of SARS-CoV-2 infection. We show that S-binding BMPCs are quiescent, which suggests that they are part of a stable compartment. Consistently, circulating resting memory B cells directed against SARS-CoV-2 S were detected in the convalescent individuals. Overall, our results indicate that mild infection with SARS-CoV-2 induces robust antigen-specific, long-lived humoral immune memory in humans.